Eastern brown snake

Eastern Brown Snake ( Pseudonaja textilis )

The Eastern Brown Snake ( Pseudonaja textilis ), also Common Brown snake is a occurring in Australia and New Guinea poison snake from the genus of brown snakes ( Pseudonaja ). Measured by their LD50 value is after also native to Australia Inlandtaipan the zweitgiftigste land snake in the world.

Occurrence

It is distributed almost throughout Australia and Queensland, the Northern Territory, South Australia, Victoria and Western Australia to meet there in New South Wales. Furthermore, the poisonous snake inhabited the north of Australia located Guinea.

Features

Physique

The Eastern Brown snake is about 1.5 m long. Some individuals, however, proven to reach a length of up to 2.4 m, so the type of the larger of the elapids can be counted. Although it is slim, but slightly wider then other species of their genus and has a rounded head.

Coloring

It is usually light brown to dark brown in color in the adult stage, which coloration varies greatly and occur also black colored copies.

Behavior

The Eastern Brown snake is considered very dangerous, as they are very nervous and aggressive responding in encounters, which causes frequent biting result. She overcomes her body into an S - shape and then bites extremely fast and usually multiple times ( 4-5 times ) in order to inject the maximum amount of poison.

Toxic effect

The venom of the Eastern Brown snake is extremely effective - the LD50 for mice is 0,041 mg per kilogram of body weight, making it one of the most potent toxins known in snakes.

The toxin comprises mainly the protein Textilinin which is derived from the Latin name of the queue. This ingredient enables the prothrombin and converts it into thrombin, which is one of the most important processes for the clotting of blood. Furthermore, it inhibits the protein plasmin, thus preventing the dissolution of the blood clot. This gives rise to a so-called consumptive coagulopathy, as all clotting factors are used up so much that the blood then is almost incoagulable.

The neurotoxic component of the toxin causes the release of acetylcholine, which is an important neurotransmitter which is fully set. This results from a failure to inhibit the spread of electrical stimulation of the nerves to the muscles, resulting in a neuromuscular blockade (muscle paralysis).